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Please use this identifier to cite or link to this item: http://scholars.ntou.edu.tw/handle/123456789/23792
DC FieldValueLanguage
dc.contributor.authorLin, Chia-Yuanen_US
dc.contributor.authorHuang, Yan-Ningen_US
dc.contributor.authorFu, Ru-Hueien_US
dc.contributor.authorLiao, Yu-Hsinen_US
dc.contributor.authorKuo, Tzu-Yuen_US
dc.contributor.authorTsai, Chia-Wenen_US
dc.date.accessioned2023-05-10T06:31:39Z-
dc.date.available2023-05-10T06:31:39Z-
dc.date.issued2021-01-
dc.identifier.issn09447113-
dc.identifier.urihttp://scholars.ntou.edu.tw/handle/123456789/23792-
dc.description.abstractImpairment of mitochondrial biogenesis is associated with the pathological progression of Parkinson's disease (PD). Parkin-interacting substrate (PARIS) can be ubiquitinated by parkin and prevents the repression of proliferator-activated receptor gamma coactivator-1-alpha (PGC-1α).en_US
dc.language.isoen_USen_US
dc.publisherELSEVIERen_US
dc.relation.ispartofPhytomedicine : international journal of phytotherapy and phytopharmacologyen_US
dc.subject6-hydroxydopamineen_US
dc.subjectCarnosic aciden_US
dc.subjectMitochondrial biogenesisen_US
dc.subjectPARISen_US
dc.subjectPGC-1αen_US
dc.titlePromotion of mitochondrial biogenesis via the regulation of PARIS and PGC-1α by parkin as a mechanism of neuroprotection by carnosic aciden_US
dc.typejournal articleen_US
dc.identifier.doi10.1016/j.phymed.2020.153369-
dc.identifier.pmid33070082-
dc.identifier.isiWOS:000592920900012-
dc.relation.journalvolume80en_US
dc.identifier.eissn1618-095X-
item.openairetypejournal article-
item.fulltextno fulltext-
item.grantfulltextnone-
item.openairecristypehttp://purl.org/coar/resource_type/c_6501-
item.cerifentitytypePublications-
item.languageiso639-1en_US-
crisitem.author.deptNational Taiwan Ocean University,NTOU-
crisitem.author.deptCollege of Life Sciences-
crisitem.author.deptDepartment of Food Science-
crisitem.author.parentorgNational Taiwan Ocean University,NTOU-
crisitem.author.parentorgCollege of Life Sciences-
Appears in Collections:食品科學系
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